Effect of the Maze Procedure for Atrial Fibrillation on Atrial and Brain Natriuretic Peptide
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Epicardial Left Atrial Appendage Exclusion Reduces Blood Pressure in Patients With Atrial Fibrillation and Hypertension
2018, Journal of the American College of CardiologyCitation Excerpt :ANP is an important neurohormonal regulator that plays a vital role in cardiovascular homeostasis by regulating natriuresis and diuresis in response to volume expansion to maintain adequate salt and water balance (10). Earlier studies in patients who underwent a maze procedure with bilateral atrial appendectomies demonstrated reduced ANP in the early post-operative phase (11,12) that remained low at 2 years (12). Despite the decrease in ANP levels, there were no reported data on its impact on systemic blood pressure until the recent observations from our group performing epicardial LAAE in patients with AF (6,7).
A novel device for left atrial appendage exclusion: The third-generation atrial exclusion device
2008, Journal of Thoracic and Cardiovascular SurgeryCitation Excerpt :Several investigators have demonstrated that the level of plasma ANP decreased after bilateral atrial appendectomy in animal models. In clinical settings, attenuation of ANP has been found in patients after the maze procedure and bilateral atrial appendectomy.20,21 This implies that amputation of both atrial appendages could affect renal function.
Success of Surgical Left Atrial Appendage Closure. Assessment by Transesophageal Echocardiography
2008, Journal of the American College of CardiologyAssociation of atrial fibrillation and amino-terminal pro-brain natriuretic peptide concentrations in dyspneic subjects with and without acute heart failure: Results from the ProBNP Investigation of Dyspnea in the Emergency Department (PRIDE) study
2007, American Heart JournalCitation Excerpt :The challenge is to understand the association between AF and the elevations of NT-proBNP seen in those patients without acute HF. Recent data suggest that BNP and NT-proBNP levels may be correlated with the presence of AF6,7,11-18 and that natriuretic peptide values may decline significantly after conversion from AF to normal sinus rhythm,19-25 suggesting a mechanism in this arrhythmia that leads to an increase in BNP or NT-proBNP. Possible mechanisms include a primary increase in natriuretic peptide production induced by AF, and/or a secondary increase due to impaired cardiac function, ventricular wall stress, or increased ventricular filling pressures without clinically overt HF.14,26