Clinical StudyImpaired left ventricular relaxation during pacing-induced ischemia*
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Cited by (214)
Deliberations on Diastolic Heart Failure
2017, American Journal of CardiologyCitation Excerpt :Early reports were directed at LV diastolic compliance in pressure overload hypertrophy, and it was suggested that the stiff hypertrophic ventricle had a limited potential to use the Frank-Starling mechanism.8 At the same time, a dramatic impact of acute ischemia on the diastolic properties of the ventricle was reported.9,10 During pacing-induced angina pectoris, a substantial increase in LV diastolic pressure was seen despite little change in diastolic volume.
Frequency of diastolic third and fourth heart sounds with myocardial ischemia induced during percutaneous coronary intervention
2009, Journal of ElectrocardiologyCitation Excerpt :Ventricular dysfunction during myocardial ischemia and infarction has been frequently observed. Investigators have demonstrated abnormal wall motion in the ischemic myocardial zone on echocardiography3 and a rise in left ventricular end-diastolic pressure (LVEDP) as measured by pulmonary artery catheter monitoring4-5 or direct measurement of ventricular pressure,6-7 reflecting reduced contractility and/or compliance of the myocardium. Also, a few animal study results8-9 suggest that, without ST changes, ventricular function may be impaired during repeated PCI balloon occlusions, inducing “stunning of the myocardium.”
Advanced Hemodynamic Assessment of Ventricular Function
2008, Textbook of Clinical HemodynamicsEvaluation of cardiac function by tissue Doppler echocardiography: Hemodynamic determinants and clinical application
2005, Ultrasound in Medicine and BiologyInspiratory muscle weakness in diastolic dysfunction
2004, ChestCitation Excerpt :The isovolumic relaxation time (IVRT) was computed and used as the echocardiographic index of diastolic function. IVRT is affected by heart rate (HR); therefore, to normalize the data, the IVRT was corrected for HR.15 Left ventricular mass was computed with the Penn convention.16
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This study was supported by North Carolina Heart Association Grant 1971-72-A-7 and National Institute of Health Grants HL 14,883-01 and HL 05727-06.
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Recipient of Special Research Fellowship 1-F03-HL-55243-01 from the National Heart and Lung Institute.